Mutant strain: LMJ.RY0402.105841

Overview

Background strain: cMJ030 Click here to order this strain from the Chlamydomonas Resource Center. The background strain comes free with every order.
Transformation condition: RY0402
Antibiotic resistance:resistant to paromomycin

Insertion junctions

Insertion junction Locus systematic id Locus common name Defline Feature Confidence (%)
LMJ.RY0402.105841_1 Cre09.g396950 MFT25,CGL15,PHT4I,PHT4-9 Na+-dependent inorganic phosphate cotransporter; (1 of 4) K08193 - MFS transporter, ACS family, solute carrier family 17 (sodium-dependent inorganic phosphate cotransporter), other (SLC17A) intron 73

If you use this mutant for your work, please cite: Li et al. 2019 Nature Genetics.

Phenotypes

This mutant exhibited a phenotype under the following conditions:

Carbon concentrating mechanism, screen 1 of 11 (~40.0% growth defect)
Carbon concentrating mechanism, screen 2 of 11 (~30.0% growth defect)
Carbon concentrating mechanism, screen 3 of 11 (~30.0% growth defect)
Carbon concentrating mechanism, screen 4 of 11 (~30.0% growth defect)
Carbon concentrating mechanism, screen 5 of 11 (~30.0% growth defect)
Carbon concentrating mechanism, screen 6 of 11 (~20.0% growth defect)
Carbon concentrating mechanism, screen 7 of 11 (~30.0% growth defect)
Carbon concentrating mechanism, screen 10 of 11 (~50.0% growth defect)
Carbon concentrating mechanism, screen 11 of 11 (~40.0% growth defect)
High temperature, screen 1 of 7 (~10.0% improved growth)
Hypoxic, screen 1 of 1 (~60.0% growth defect)
Hypoxic and high light, screen 1 of 1 (~70.0% growth defect)
Photosynthesis, screen 1 of 5 (~50.0% growth defect)
Photosynthesis, screen 4 of 5 (~50.0% growth defect)
Photosynthesis, screen 5 of 5 (~40.0% growth defect)

Note: Not all phenotypes can be confidently associated with a specific gene or insertion; phenotypes can be caused by unmapped second-site mutations. Please see the gene pages for statistically significant gene-phenotype links. These data are from a pooled phenotyping experiment.

If you reference the phenotypic data above in a manuscript, please cite: Fauser et al. 2022 Nature Genetics.