Background strain: | cMJ030 | Click here to order this strain from the Chlamydomonas Resource Center. The background strain comes free with every order. |
Transformation condition: | RY0402 | |
Antibiotic resistance: | resistant to paromomycin |
Insertion junctions | |||||
Insertion junction | Locus systematic id | Locus common name | Defline | Feature | Confidence (%) |
---|---|---|---|---|---|
LMJ.RY0402.210584_1 | Cre03.g175600 | (1 of 1) PTHR26392//PTHR26392:SF78 - MITOGEN-ACTIVATED PROTEIN KINASE KINASE KINASE 7-RELATED // SUBFAMILY NOT NAMED | 3'UTR | 95 | |
LMJ.RY0402.210584_2 | Cre03.g175600 | (1 of 1) PTHR26392//PTHR26392:SF78 - MITOGEN-ACTIVATED PROTEIN KINASE KINASE KINASE 7-RELATED // SUBFAMILY NOT NAMED | 3'UTR | 95 | |
LMJ.RY0402.210584_3 | Cre17.g741500 | KIL7 | Putative kinesin-like motor protein; (1 of 1) 3.4.21.72//3.6.4.4 - IgA-specific serine endopeptidase / Immunoglobulin A1 protease // Plus-end-directed kinesin ATPase / Kinesin | CDS | 73 |
If you use this mutant for your work, please cite: Li et al. 2019 Nature Genetics.
This mutant exhibited a phenotype under the following conditions:
Carbon concentrating mechanism, screen 8 of 11 (~20.0% growth defect) |
Note: Not all phenotypes can be confidently associated with a specific gene or insertion; phenotypes can be caused by unmapped second-site mutations. Please see the gene pages for statistically significant gene-phenotype links. These data are from a pooled phenotyping experiment.
If you reference the phenotypic data above in a manuscript, please cite: Fauser et al. 2022 Nature Genetics.