| Background strain: | cMJ030 | Click here to order this strain from the Chlamydomonas Resource Center. The background strain comes free with every order. |
| Transformation condition: | RY0402 | |
| Antibiotic resistance: | resistant to paromomycin |
Insertion junctions | |||||
| Insertion junction | Locus systematic id | Locus common name | Defline | Feature | Confidence (%) |
|---|---|---|---|---|---|
| LMJ.RY0402.221749_1 | Cre02.g113350 | Protein tyrosine kinase; (1 of 1) IPR000719//IPR001245//IPR011009//IPR020635 - Protein kinase domain // Serine-threonine/tyrosine-protein kinase catalytic domain // Protein kinase-like domain // Tyrosine-protein kinase, catalytic domain | 3'UTR | 73 | |
| LMJ.RY0402.221749_2 | Cre05.g241635 | (1 of 1) IPR012344//IPR014001//IPR027417 - Matrix protein, lentiviral and alpha-retroviral // Helicase superfamily 1/2, ATP-binding domain // P-loop containing nucleoside triphosphate hydrolase | intron | 73 | |
If you use this mutant for your work, please cite: Li et al. 2019 Nature Genetics.
This mutant exhibited a phenotype under the following conditions:
| Paromomycin, screen 1 of 6 (~50.0% growth defect) |
| Paromomycin, screen 3 of 6 (~70.0% growth defect) |
Note: Not all phenotypes can be confidently associated with a specific gene or insertion; phenotypes can be caused by unmapped second-site mutations. Please see the gene pages for statistically significant gene-phenotype links. These data are from a pooled phenotyping experiment.
If you reference the phenotypic data above in a manuscript, please cite: Fauser et al. 2022 Nature Genetics.