| Background strain: | cMJ030 | Click here to order this strain from the Chlamydomonas Resource Center. The background strain comes free with every order. |
| Transformation condition: | RY0402 | |
| Antibiotic resistance: | resistant to paromomycin |
Insertion junctions | |||||
| Insertion junction | Locus systematic id | Locus common name | Defline | Feature | Confidence (%) |
|---|---|---|---|---|---|
| LMJ.RY0402.229313_1 | Cre01.g034900 | CDPK5 | (1 of 4) PTHR24347:SF138 - PROTEIN F32D8.1; Calcium/calmodulin-dependent protein kinase | intron | 95 |
| LMJ.RY0402.229313_2 | Cre01.g034900 | CDPK5 | (1 of 4) PTHR24347:SF138 - PROTEIN F32D8.1; Calcium/calmodulin-dependent protein kinase | intron | 95 |
| LMJ.RY0402.229313_3 | Cre06.g259100 | POB30 | Proteome of basal body 30 | intron | 95 |
| LMJ.RY0402.229313_4 | Cre06.g259100 | POB30 | Proteome of basal body 30 | intron | 95 |
If you use this mutant for your work, please cite: Li et al. 2019 Nature Genetics.
This mutant exhibited a phenotype under the following conditions:
| Carbon concentrating mechanism, screen 1 of 11 (~60.0% growth defect) |
| Carbon concentrating mechanism, screen 3 of 11 (~80.0% growth defect) |
| Carbon concentrating mechanism, screen 4 of 11 (~80.0% growth defect) |
| Carbon concentrating mechanism, screen 5 of 11 (~60.0% growth defect) |
| Carbon concentrating mechanism, screen 6 of 11 (~60.0% growth defect) |
| Carbon concentrating mechanism, screen 7 of 11 (~60.0% growth defect) |
| Carbon concentrating mechanism, screen 9 of 11 (~80.0% growth defect) |
| Carbon concentrating mechanism, screen 11 of 11 (~80.0% growth defect) |
| Photosynthesis, screen 3 of 5 (~80.0% growth defect) |
| Photosynthesis, screen 5 of 5 (~80.0% growth defect) |
Note: Not all phenotypes can be confidently associated with a specific gene or insertion; phenotypes can be caused by unmapped second-site mutations. Please see the gene pages for statistically significant gene-phenotype links. These data are from a pooled phenotyping experiment.
If you reference the phenotypic data above in a manuscript, please cite: Fauser et al. 2022 Nature Genetics.